×
Home
Archive Submission Guidelines
News Contact
Review article
Crossmark

Helicobacter Pylori Infection and Upper Gastrointestinal Disease

By
Marina Dinić ,
Marina Dinić
Dobrila Đorđević ,
Dobrila Đorđević
Gordana Tasić ,
Gordana Tasić
Branislava Kocić ,
Branislava Kocić
Milena Bogdanović
Milena Bogdanović

Abstract

Helicobacter pylori infection is one of the most common bacterial infections in humans, being present in over a half of the world population. Helicobacter pylori infection, by itself, does not necessarily produce the symptoms of gastrointestinal tract diseases, but certainly presents a risk for their development. The clinical outcome of Helicobacter pylori infection depends on the interaction of numerous factors: the virulence of a bacterial strain, genetic predisposition and premorbid host conditions, as well as the environmental factors. Accordingly, a diagnosis of Helicobacter pylori infection will be of clinical relevance only if it is necessary to establish the cause of a disease associated with this infection. Thus, a thorough knowledge of the diseases associated with Helicobacter pylori infection is a key factor in any relevant assessment of the need for eradication therapy.

References

1.
Dinić M, Tasić G, Stanković-Dordević D, Otašević L, Tasić M, Karanikolic A. Serum anti-Helicobacter pylori IgA and IgG antibodies in asymptomatic children in Serbia. Scandinavian Journal of Infectious Diseases. 2007;39(4):303–7.
2.
Molinari M, Salio M, Galli C, Norais N, Rappuoli R, Lanzavecchia A, et al. Selective Inhibition of Ii-dependent Antigen Presentation by Helicobacter pylori Toxin VacA. The Journal of Experimental Medicine. 1998;187(1):135–40.
3.
Yamaoka Y, Kodama T, Gutierrez O, Kim JG, Kashima K, Graham DY. Relationship between            Helicobacter pylori iceA            ,            cagA            , and            vacA            Status and Clinical Outcome: Studies in Four Different Countries. Journal of Clinical Microbiology. 1999;37(7):2274–9.
4.
Smoot DT, Mobley HL, Chippendale GR, Lewison JF, Resau JH. Helicobacter pylori urease activity is toxic to human gastric epithelial cells. Infection and Immunity. 1990;58(6):1992–4.
5.
Gerhard M, Lehn N, Neumayer N, Borén T, Rad R, Schepp W, et al. Clinical relevance of the            Helicobacter pylori            gene for blood-group antigen-binding adhesin. Proceedings of the National Academy of Sciences. 1999;96(22):12778–83.

Citation

Article metrics

Google scholar: See link

The statements, opinions and data contained in the journal are solely those of the individual authors and contributors and not of the publisher and the editor(s). We stay neutral with regard to jurisdictional claims in published maps and institutional affiliations.