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EFFECT OF HALOGENATED BOROXINE ON HEMATOLOGICAL PARAMETERS IN RATS AND ON CREATINE KINASE ACTIVITY IN VITRO RETROSPECTIVE REAL-WORLD ANALYSIS

By
Safija Herenda Orcid logo ,
Safija Herenda
Contact Safija Herenda

Department of Chemistry, Faculty of Science, University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Elma Hasković Orcid logo ,
Elma Hasković

Sarajevo, Krka , Sarajevo , Bosnia and Herzegovina

Muhamed Fočak Orcid logo ,
Muhamed Fočak

Department of Biology, Faculty of Science, University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Alem Bekan Orcid logo ,
Alem Bekan

Department of Biology, Faculty of Science, University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Sabina Prevljak Orcid logo ,
Sabina Prevljak

Organizational unit clinic for radiology, Clinical Center University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Nenad Vanis Orcid logo ,
Nenad Vanis

Sarajevo, ASA Hospital Bosnia and Herzegovina

Mirza Halimić Orcid logo ,
Mirza Halimić

Pediatric Clinic, Clinical Center University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Alen Džubur Orcid logo ,
Alen Džubur

Clinic for Heart, Blood Vessel and Rheumatic Diseases, Clinical Center University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Edhem Hasković Orcid logo
Edhem Hasković

Department of Biology, Faculty of Science, University of Sarajevo , Sarajevo , Bosnia and Herzegovina

Abstract

Halogenated boroxine is an important enzyme inhibitor with potential applications in cancer therapy. This study aimed to evaluate its effects on hematological parameters in rats in vivo and creatine kinase activity in vitro. Halogenated boroxine, administered either intraperitoneally (IP) or orally, induced route-dependent changes in hematological parameters compared with controls. IP administration significantly increased neutrophils, mean corpuscular hemoglobin (MCH), and mean corpuscular hemoglobin concentration (MCHC), while decreasing hematocrit (HCT). In contrast, oral administration significantly decreased lymphocytes and increased basophils, while other hematological parameters remained unchanged. These effects were more pronounced following IP administration. In vitro, halogenated boroxine exhibited competitive inhibition of creatine kinase without pre-incubation, whereas after a 30-minute pre-incubation, the inhibition pattern indicated a non-competitive mechanism. Further analysis showed a time-dependent shift in creatine kinase inhibition mechanism following pre-incubation, characterized by a decrease in maximum reaction velocity (Vmax), while the Michaelis constant (Km) remained unaltered. These findings show that halogenated boroxine modulates specific hematological parameters and inhibits creatine kinase activity, with effects depending on the route of administration and experimental conditions.

Author Contributions

Conceptualization, S.H. and E.H.; Methodology, S.H. and E.H.; Writing – original draft, S.H., M.F., A.B., S.P., M.H., A.D. and E.H.; Writing – review & editing, S.H., N.V. and A.D.; Formal Analysis, E.H., M.F. and A.B.; Investigation, E.H., M.F. and A.B.; Supervision, S.P. and M.H. All authors have read and agreed to the published version of the manuscript.

Conflict of Interest

The authors declare no relevant conflicts of interest.

Funding Statement

Research reported in this paper was supported by Federal Ministry of Education and Science, Bosnia and Herzegovina under award number 05-35-3573-1/25.

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